机构:[1]Key Laboratory of Orthopaedics & Traumatology, Lingnan Medical Research Center, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong 510000, China.[2]Department of Orthopaedics & Traumatology, Faculty of Medicine, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR 999077, China.[3]Departments of Diagnostics of Traditional Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong 510000, China.[4]Orthopaedic Research Lab, Affiliated Hospital of Guangdong Medical University, Guangdong Medical University, Zhanjiang, Guangdong 524001, China.
National Natural Science
Foundation of China (No. 81871778, 81874000, 82272505).
语种:
外文
PubmedID:
中科院(CAS)分区:
出版当年[2023]版:
大类|2 区医学
小类|2 区生化与分子生物学2 区遗传学
最新[2025]版:
大类|2 区医学
小类|2 区生化与分子生物学2 区遗传学
第一作者:
第一作者机构:[1]Key Laboratory of Orthopaedics & Traumatology, Lingnan Medical Research Center, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong 510000, China.
共同第一作者:
通讯作者:
通讯机构:[1]Key Laboratory of Orthopaedics & Traumatology, Lingnan Medical Research Center, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong 510000, China.[2]Department of Orthopaedics & Traumatology, Faculty of Medicine, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR 999077, China.[4]Orthopaedic Research Lab, Affiliated Hospital of Guangdong Medical University, Guangdong Medical University, Zhanjiang, Guangdong 524001, China.[*1]The First Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong 510000, China.[*2]Department of Orthopaedics & Traumatology, Faculty of Medicine, The Chinese University of Hong Kong, Shatin, Hong Kong SAR 999077, China.
推荐引用方式(GB/T 7714):
Li Ying,Ding Lingli,Zhang Yage,et al.Histone demethylase Kdm5c regulates osteogenesis and bone formation via PI3K/Akt/HIF1α and Wnt/β-catenin signaling pathways[J].Genes & diseases.2024,11(1):64-66.doi:10.1016/j.gendis.2023.02.041.
APA:
Li Ying,Ding Lingli,Zhang Yage,Zhou Bingyu,Wai Man Gene Chi...&Xu Liangliang.(2024).Histone demethylase Kdm5c regulates osteogenesis and bone formation via PI3K/Akt/HIF1α and Wnt/β-catenin signaling pathways.Genes & diseases,11,(1)
MLA:
Li Ying,et al."Histone demethylase Kdm5c regulates osteogenesis and bone formation via PI3K/Akt/HIF1α and Wnt/β-catenin signaling pathways".Genes & diseases 11..1(2024):64-66